Overexpression of UCP4 in astrocytic mitochondria prevents multilevel dysfunctions in a mouse model of Alzheimer s disease
Nadia Rosenberg, Maria Reva, Francesca Binda, Leonardo Restivo, Pauline Depierre, Julien Puyal, Marc Briquet, Yann Bernardinelli, Anne‐Bérengère Rocher, Henry Markram, Jean‐Yves Chatton
This study shows that overexpressing UCP4 in astrocytic mitochondria prevents metabolic and structural neuronal dysfunctions in a mouse model of Alzheimer's disease.
Glia · Dec 20, 2022Peer-reviewedNeuroscience