Author

R. L. Smoot

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Assay:CITE-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:mouse· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

B. Li· 1 shared E. Loeuillard· 1 shared F. Rakhshan Rohakhtar· 1 shared H. Dong· 1 shared H. E. Stumpf· 1 shared J. L. Tomlinson· 1 shared J. R. Willhite· 1 shared J. Wang· 1 shared

Activity by Year

2023-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

B. Li

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1 paper

Recent·Most cited

Noncanonical TRAIL Signaling Promotes Myeloid-Derived Suppressor Cell Abundance and Tumor Progression in Cholangiocarcinoma

E. Loeuillard, B. Li, H. E. Stumpf, J. Yang, J. R. Willhite, J. L. Tomlinson, J. Wang, F. Rakhshan Rohakhtar, V. A. Simon, R. P. Graham, R. L. Smoot, H. Dong, S. I. Ilyas

This study shows that TRAIL+ cancer cells use noncanonical TRAIL signaling in MDSCs to promote their abundance and tumor progression in cholangiocarcinoma.

bioRxiv · May 24, 2023 · PreprintCancer Biology