Author

S. Camilleri-Broet

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Assay:proteomics· 3 Research Focus:cell type annotation· 2 Approach:deep learning· 2 Research Focus:differential expression· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:cell-cell communication· 1 Research Focus:clustering· 1

Frequent Co-Authors

B. Schilling· 2 shared J. Bons· 2 shared J. Rose· 2 shared N. Basisty· 2 shared P. Gascard· 2 shared P.-O. Fiset· 2 shared S. Shah· 2 shared T. D. Tlsty· 2 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 44 mutual co-author links

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2 shared papersCancer Biology

B. Schilling

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3 papers

Recent·Most cited

Concerted epithelial and stromal changes during progression of Barrett's Esophagus to invasive adenocarcinoma exposed by multi-scale, multi-omics analysis

M. Strasser, D. L. Gibbs, P. Gascard, J. Bons, J. W. Hickey, C. M. Schürch, Y. Tan, S. Black, P. Chu, A. Ozkan, N. Basisty, V. Sangwan, J. Rose, S. Shah, S. Camilleri-Broet, , L. L. Ferri

This study uses multi-omics analysis to show coordinated changes in epithelial, stromal, and immune compartments during Barrett's Esophagus progression to adenocarcinoma, revealing early appearance of cancer-associated fibroblasts and immunosuppressive microenvironment.

bioRxiv · Jun 8, 2023 · PreprintCancer Biology

Data-Independent Acquisition and Quantification of Extracellular Matrix from Human Lung in Chronic Inflammation-Associated Carcinomas

J. Bons, D. Pan, S. Shah, R. Bai, C. Chen-Tanyolac, X. Wang, D. R. Fels Elliott, A. Urisman, A. O'Broin, N. Basisty, J. Rose, V. Sangwan, S. Camilleri-Broet, J. Tankel, P. Gascard, , B. Schilling

The paper develops a proteomic pipeline to analyze ECM remodeling in lung squamous cell carcinoma, identifying 529 differentially expressed proteins and potential biomarkers.

bioRxiv · Aug 5, 2022Cancer Biology

Gram-negative bacterial infection increases lung cancer metastasis via Toll-like receptor 1 activation and increased cancer cell proliferation post-tumor adhesion

R. F. Rayes, M. G. Wilson, S. D. Gowing, F. Bourdeau, B. Giannias, S. Camilleri-Broet, P.-O. Fiset, S. Najmeh, J. D. Spicer, L. E. Ferri, J. Cools-Lartigue

This study shows that gram-negative bacterial infection increases lung cancer metastasis through TLR4 activation and enhanced cancer cell proliferation after tumor adhesion.

bioRxiv · Sep 15, 2021 · PreprintCancer Biology