MAFB surrogates the glucocorticoid receptor ability to induce tolerogenesis in dendritic cells
O. Morante-Palacios, L. Ciudad, R. Micheroli, C. de la Calle-Fabregat, T. Li, G. Barbisan, M. Houtman, S. Edalat, M. Frank-Bertoncelj, C. Ospelt, E. Ballestar
This study identifies MAFB as a key mediator of GC-induced tolerogenesis in dendritic cells, showing its role in epigenomic and transcriptomic remodeling.
bioRxiv · Jul 27, 2021 · PreprintImmunology