Author

S. Z. Sheikh

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Research Focus:differential expression· 2 Organism:human· 2 Organism:mouse· 2 Approach:statistical model· 2 Assay:ATAC-seq· 1

Frequent Co-Authors

B. P. Keith· 2 shared E. C. Steinbach· 2 shared T. S. Furey· 2 shared T. Toyonaga· 2 shared A. Berglind· 1 shared A. E. Berglind· 1 shared A. W. Burks· 1 shared C. Beasley· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersImmunology

B. P. Keith

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

BET protein inhibition regulates macrophage chromatin accessibility and microbiota-dependent colitis

M. Hoffner O'Connor, A. Berglind, M. M. Kennedy, B. P. Keith, Z. J. Lynch, M. R. Schaner, E. C. Steinbach, J. Herzog, O. K. Trad, W. R. Jeck, J. C. Arthur, R. B. Sartor, T. Furey, S. Z. Sheikh

This study shows that BET protein inhibition reduces macrophage inflammation and colitis severity by altering chromatin accessibility and gene expression.

bioRxiv · Jul 15, 2021 · PreprintImmunology

Increased intestinal permeability in an orally-reactive peanut allergy model identifies Angiopoietin like-4 as a biomarker

E. C. Steinbach, J. M. Smeekens, S. Roy, T. Toyonaga, C. Cornaby, L. B. Perini, A. E. Berglind, M. D. Kulis, E. H. Kim, M. T. Ferris, T. S. Furey, A. W. Burks, S. Z. Sheikh

The paper identifies Angptl4 as a biomarker of peanut allergy by showing increased intestinal permeability and serum Angptl4 levels in a mouse model and pediatric patients.

bioRxiv · Jul 14, 2021 · PreprintCell Biology

Decreased Colonic Activin Receptor Like Kinase 1 Disrupts Epithelial Barrier integrity and is associated with a poor clinical outcome in Crohns disease

T. Toyonaga, B. P. Keith, J. Barrow, M. S. Schaner, E. A. Wolber, C. Beasley, J. Huling, Y. Wang, N. L. Allbritton, N. Chaumont, T. S. Sadiq, M. J. Koruda, R. Rahbar, T. S. Furey, P. Sethupathy, S. Z. Sheikh

This study identifies ALK1 as a target of miR-31-5p in CD, showing that decreased ALK1 disrupts epithelial barrier integrity and correlates with poor clinical outcomes.

bioRxiv · Feb 21, 2020Cell Biology