Author

Stephen J Royle

Primary ORCID profile (0000-0001-8927-6967 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

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Same name, identified by ORCID

Stephen J RoyleiD1 paper · University of Warwick

Biology Disciplines

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:protein folding & dynamics· 1 Assay:proteomics· 1

Frequent Co-Authors

Andrew B Fielding· 1 shared Anna K Willox· 1 shared Cristina Gutiérrez-Caballero· 1 shared Gisela Gassner· 1 shared Nicholas J Carter· 1 shared Richard Bayliss· 1 shared Satdip Kaur· 1 shared Selena G Burgess· 1 shared

Activity by Year

2014-2014 · peak 3/yr

Co-Author Collaboration Network

9 top collaborators · 10 mutual co-author links

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1 shared paperCell Biology

Andrew B Fielding

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3 papers

Recent·Most cited

TACC3-ch-TOG track the growing tips of microtubules independently of clathrin and Aurora-A phosphorylation

Cristina Gutiérrez-Caballero, Selena G Burgess, Richard Bayliss, Stephen J Royle

This study identifies a TACC3-ch-TOG pool independent of clathrin and Aurora-A phosphorylation that tracks microtubule plus-ends, revealing distinct subcellular pools of TACC3, ch-TOG, and clathrin.

bioRxiv · Nov 4, 2014Cell Biology

Non-specificity of Pitstop 2 in clathrin-mediated endocytosis

Anna K Willox, Yasmina M.E. Sahraoui, Stephen J Royle

This study shows that Pitstop 2 inhibits clathrin-mediated endocytosis non-specifically, not through its intended mechanism, and warns against using it to infer the function of clathrin's N-terminal domain.

Biology Open · Apr 4, 2014Peer-reviewedCell Biology

An unmet actin requirement explains the mitotic inhibition of clathrin-mediated endocytosis

Satdip Kaur, Andrew B Fielding, Gisela Gassner, Nicholas J Carter, Stephen J Royle

This study identifies an unmet actin requirement as the cause of mitotic inhibition of clathrin-mediated endocytosis, showing that restoring actin availability can restart CME in mitotic cells.

bioRxiv · Feb 18, 2014Cell Biology