Lamin A/C Deficiency Enables Increased Myosin2 Bipolar Filament Ensembles Which Promote Divergent Actomyosin Network Anomalies Through Self Organization
O'Neil Wiggan, Jennifer G DeLuca, Timothy J Stasevich, James R Bamburg
This study shows that lamin A/C deficiency leads to increased myosin-II bipolar filament ensembles and actomyosin network anomalies through self-organization mechanisms.
bioRxiv · Jul 10, 2020Cell Biology