Author

V. Prahlad

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Organism:C. elegans· 3 Assay:DNA methylation· 3 Approach:experimental assay· 3 Assay:ChIP-seq· 2 Research Focus:chromatin & epigenetic regulation· 2 Research Focus:development & morphogenesis· 2 Research Focus:gene regulatory network inference· 2 Approach:statistical model· 2

Frequent Co-Authors

F. K. Ooi· 2 shared S. Das· 2 shared J. A. Weiner· 1 shared J. Cruz Corchado· 1 shared L. C. C. Fuller· 1 shared S. Min· 1 shared

Activity by Year

2017-2021 · peak 1/yr

Co-Author Collaboration Network

6 top collaborators · 11 mutual co-author links

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2 shared papersCell Biology

F. K. Ooi

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Germline activity of the heat shock factor HSF-1 programs the insulin-receptor daf-2 in C. elegans

V. Prahlad, S. Das, S. Min

HSF-1 recruits MET-2 to increase H3K9me2 levels at daf-2, silencing the insulin-like receptor and enhancing offspring stress resilience in C. elegans.

bioRxiv · Feb 22, 2021Cell Biology

Serotonin signaling by maternal neurons upon stress ensures progeny survival

S. Das, F. K. Ooi, J. Cruz Corchado, L. C. C. Fuller, J. A. Weiner, V. Prahlad

This study shows maternal serotonin signaling during stress in C. elegans protects offspring by enabling HSF1 to alter chromatin in germ cells, ensuring their viability and stress tolerance.

bioRxiv · Jan 20, 2020Cell Biology

Olfactory learning primes the heat shock transcription factor HSF-1 to enhance the expression of molecular chaperone genes in C. elegans

F. K. Ooi, V. Prahlad

This study shows that olfactory learning in C. elegans primes HSF-1 to enhance chaperone gene expression, improving survival upon pathogen exposure through serotonin-dependent mechanisms.

bioRxiv · Aug 8, 2017Cell Biology