Author

W. M. Kuebler

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2 Biochemistry· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:statistical model· 2 Approach:deep learning· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1

Frequent Co-Authors

A. Abukiwan· 1 shared A. Birnhuber· 1 shared A. C. Hocke· 1 shared A. L. Olivares· 1 shared A. Weigert· 1 shared A.-S. Schultz· 1 shared B. Seeliger· 1 shared C. De La Torre· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperPhysiology

A. Abukiwan

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Author profile →

3 papers

Recent·Most cited

Inference of alveolar capillary network connectivity from blood flow dynamics

K. Schmid, A. L. Olivares, O. Camara, W. M. Kuebler, M. Ochs, A. C. Hocke, S. C. Fischer

This paper infers the connectivity of alveolar capillary networks from blood flow dynamics using computational models and simulations.

bioRxiv · Jan 22, 2024Physiology

NFAT5-dependent transcriptional stress control of endothelial cells prevents maladaptive remodeling of pulmonary arterioles in the hypoxic lung

H. Laban, S. Siegmund, K. Schlereth, L. Brandenburg, F. A. Trogisch, A. Weigert, C. De La Torre, A. Abukiwan, C. Mogler, M. Hecker, W. M. Kuebler, T. Korff

This study shows that NFAT5 in endothelial cells limits hypoxia-induced maladaptive remodeling of pulmonary arterioles by restricting HIF1-mediated Pdgfb expression.

bioRxiv · Oct 18, 2023Physiology

Altered fibrin clot structure contributes to thrombosis risk in severe COVID-19

M. Wygrecka, A. Birnhuber, B. Seeliger, L. Michalick, O. Pak, A.-S. Schultz, F. Schramm, M. Zacharias, G. Gorkiewicz, S. David, T. Welte, J. J. Schmidt, N. Weissmann, R. T. Schermuly, G. Barreto, , K. T. Preissner

The paper shows that altered fibrin clot structure in severe COVID-19 patients contributes to thrombosis risk by forming compact, fibrinolysis-resistant clots.

bioRxiv · Sep 17, 2021Biochemistry