Author

Vasiliki Triantafyllia

Biomedical Research Foundation of the Academy of AthensORCID 0000-0001-5832-4014Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetic & Genomic Medicine· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Assay:metabolomics· 1

Frequent Co-Authors

Eleni SioutiiD· 2 shared Evangelos AndreakosiD· 2 shared Abdul Rouf BandayiD· 1 shared Adam J. GehringiD· 1 shared Alicia K. SmithiD· 1 shared Amy A HutchinsoniD· 1 shared Andrea N. Burnett‐HartmaniD· 1 shared Anurag VermaiD· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersImmunology

Eleni SioutiORCID

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2 papers

Recent·Most cited

ImmunologyProceedings of the National Academy of SciencesPeer-reviewedJun 1, 2026

Polyunsaturated fatty acid-derived lipid mediator networks characterize COVID-19 severity and risk for critical illness

Maria Papadaki, Eleftherios Pavlos, Marc Dubourdeau…·Evangelos Andreakos

The paper identifies lipid mediator networks associated with COVID-19 severity and risk of critical illness using multi-omics profiling across disease severity groups.

1 citation

humanbulk RNA-seq

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Genetic & Genomic MedicineNature GeneticsPeer-reviewedJul 14, 2022

Genetic regulation of OAS1 nonsense-mediated decay underlies association with risk of severe COVID-19

Abdul Rouf Banday, Megan Lynn Stanifer, Oscar Flórez-Vargas…·Ludmila Prokunina‐Olsson

The paper identifies a Neandertal introgressed haplotype in the OAS1 gene associated with increased risk of severe COVID-19 and shows it impairs SARS-CoV-2 clearance through genetic regulation of OAS1 expression.

130 citationstop 1%

humanwhole-genome sequencing

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