Author

Emmanuel Deau

ORCID 0000-0001-6299-9946Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Organism:human· 1 Organism:mouse· 1 Organism:rat· 1

Frequent Co-Authors

Laurent MeijeriD· 2 shared Alexander J Eichert· 1 shared Alexander Young· 1 shared Ambrosius P. SnijdersiD· 1 shared Bryana N. Harris· 1 shared Bryce Cameron MurilloiD· 1 shared Callan L BuechsenschuetziD· 1 shared Catherine Zhao· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersPharmacology & Toxicology

Laurent MeijerORCID

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2 papers

Recent·Most cited

Pharmacology & ToxicologyJCI InsightPeer-reviewedSep 17, 2026

Network Modeling Predicts How DYRK1A Inhibition Promotes Cardiomyocyte Cycling after Ischemic/Reperfusion Injury

Bryce Cameron Murillo, Alexander Young, Kaitlyn L. Wintruba…·Matthew J. Wolf

The paper develops a computational network model predicting DYRK1A inhibition promotes cardiomyocyte cycling after ischemic/reperfusion injury, validated through experiments in neonatal rat cardiomyocytes.

2 citations

ratbulk RNA-seq

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Developmental BiologybioRxivPreprintSep 18, 2023

Congenital heart defects in Down syndrome are caused by increased dosage of DYRK1A

Eva Lana‐Elola, Rifdat Aoidi, Miriam Llorian…·Victor L. J. Tybulewicz

The paper shows increased DYRK1A dosage causes mitochondrial dysfunction and congenital heart defects in Down syndrome, which can be mitigated by a DYRK1A inhibitor.

DOI 10.1101/2023.09.18.558244

humanmouse

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