Author

Jie Ling

Central South UniversityORCID 0000-0001-7054-5119Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Jie Ling1 paper · Sun Yat-sen University

Biology Disciplines

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Developmental Biology· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Assay:ATAC-seq· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:differential expression· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Ah Reum KimiD· 1 shared Aida Nourbakhsh· 1 shared Bong Jik KimiD· 1 shared Byung Yoon ChoiiD· 1 shared Changjoon Justin LeeiD· 1 shared Chufeng HeiD· 1 shared Dong Ho WooiD· 1 shared Doo‐Yi OhiD· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperGenetics

Ah Reum KimORCID

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2 papers

Recent·Most cited

Developmental BiologybioRxivPreprintSep 24, 2024

Rfx3 controls outer hair cell differentiation, maintenance, and hair bundle formation by regulating the expression of Insm1, Ikzf2, and Triobp genes

Penghui Zhang, Yafan Wang, Xiang Guo…·Jun Li

This study identifies Rfx3's role in outer hair cell development by regulating Insm1, Ikzf2, and Triobp through transcriptional activation and repression.

1 citation

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GeneticsProceedings of the National Academy of SciencesPeer-reviewedMay 28, 2021

Mutations in TMEM43 cause autosomal dominant auditory neuropathy spectrum disorder via interaction with Connexin-mediated passive conductance channels

Minwoo Wendy Jang, Doo‐Yi Oh, Eunyoung Yi…·Byung Yoon Choi

This study identifies TMEM43 mutations as a cause of autosomal dominant auditory neuropathy spectrum disorder by showing their interaction with Connexin-mediated channels in cochlear glia-like supporting cells.

33 citations

humanexperimental assay

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