Ulf Hedin

This study identifies MEIS1 as an activator of myocardin and vascular smooth muscle cell differentiation, showing its role in promoting the quiescent, differentiated state.

This study shows that FcRn-mediated antibody recycling promotes atherosclerotic plaque vulnerability by enhancing inflammation and matrix degradation.

This study identifies EV-driven endothelial communication in human carotid atherosclerosis, showing increased angiogenic activity in symptomatic plaques.

This study identifies FHL5 as a causal gene for vascular disease risk through eQTL colocalization and functional analyses, showing its role in vascular remodeling and calcification.