NeurosciencebioRxivPreprint
Binding of Filamentous Actin to CaMKII as a Potential Mechanism for the Regulation of Bidirectional Synaptic Plasticity by betaCaMKII in Cerebellar Purkinje Cells
Thiago M. Pinto, Maria J. Schilstra, Antônio C. Roque, Volker Steuber
The paper develops a computational model to show that betaCaMKII regulates bidirectional synaptic plasticity in cerebellar Purkinje cells through actin binding and AMPA receptor phosphorylation.
DOI 10.1101/521542