Thorsten U. Vogel

The study shows that cross-reactive antibodies, not cross-inhibiting ones, are responsible for broad protection against heterologous HxN2 influenza viruses.

This paper describes the generation of a mouse-adapted influenza B virus strain that gains pathogenicity in mice through mutations in non-HA/NA gene segments, independent of hemagglutinin and neuraminidase.

The paper evaluates the antigenic evolution of N1 neuraminidase in H1N1 influenza viruses from 2009 to 2020, identifying key mutations driving antigenic changes and their impact on antibody neutralization.