Author

Heather K. Le Bleu

University of OregonORCID 0000-0002-0672-9076Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:zebrafish· 4 Approach:experimental assay· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Assay:imaging· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Astra L. HenneriD· 4 shared Kryn StankunasiD· 4 shared Amy E. RobbinsiD· 2 shared Gabriel A. YetteiD· 2 shared Joshua A. BraunsteiniD· 2 shared Scott Stewart· 2 shared Scott StewartiD· 2 shared Victor M. LewisiD· 2 shared

Activity by Year

2019-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 34 mutual co-author links

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4 shared papersDevelopmental Biology

Astra L. HennerORCID

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4 papers

Recent·Most cited

Voltage-gated calcium channels generate blastema Ca2+ fluxes restraining zebrafish fin regenerative outgrowth

Heather K. Le Bleu, Rea G. Kioussi, Astra L. Henner, Victor M. Lewis, Scott Stewart, Kryn Stankunas

The paper identifies voltage-gated calcium channels as regulators of zebrafish fin regeneration, showing their role in controlling outgrowth scale through Ca2+ fluxes.

Development · Dec 1, 2025Peer-reviewedDevelopmental Biology

Insulin-like growth factor receptor / mTOR signaling elevates global translation to accelerate zebrafish fin regenerative outgrowth

Victor M. Lewis, Heather K. Le Bleu, Astra L. Henner, Hannah Markovic, Amy E. Robbins, Scott Stewart, Kryn Stankunas

The paper identifies IGFR/mTOR signaling as a driver of increased translation during zebrafish fin regeneration, accelerating outgrowth through coordinated cell behavior.

Developmental Biology · Jun 7, 2023Peer-reviewedDevelopmental Biology

longfin causes cis-ectopic expression of the kcnh2a ether-a-go-go K+ channel to autonomously prolong fin outgrowth

Scott Stewart, Heather K. Le Bleu, Gabriel A. Yette, Astra L. Henner, Amy E. Robbins, Joshua A. Braunstein, Kryn Stankunas

The paper shows that the longfint2 mutation causes overexpression of kcnh2a, leading to prolonged fin outgrowth through ion signaling in the mesenchymal lineage.

Development · Apr 26, 2021Peer-reviewedDevelopmental Biology

Skeletal geometry and niche transitions restore organ size and shape during zebrafish fin regeneration

Scott Stewart, Gabriel A. Yette, Heather K. Le Bleu, Astra L. Henner, Joshua A. Braunstein, Jad W. Chehab, Michael J. Harms, Kryn Stankunas

The paper shows that zebrafish fin regeneration size and shape depend on niche cells and skeletal geometry, not cell memories.

bioRxiv · Oct 2, 2019 · PreprintDevelopmental Biology