Author

David W. Clark

University of EdinburghORCID 0000-0002-1025-9185Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genomics· 2 Genetics· 1

Signature Assays, Organisms & Methods

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Research Focus:GWAS / association mapping· 3 Organism:human· 3 Approach:statistical model· 3 Assay:GWAS summary statistics· 2 Assay:proteomics· 1

Frequent Co-Authors

James F. WilsoniD· 3 shared Peter K. JoshiiD· 3 shared Andrew D. BretherickiD· 2 shared Paul R. H. J. TimmersiD· 2 shared Tõnu EskoiD· 2 shared Aaron Kleinman· 1 shared Abdel AbdellaouiiD· 1 shared Abraham A. PalmeriD· 1 shared

Activity by Year

2018-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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3 shared papersGenomics

James F. WilsonORCID

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3 papers

Recent·Most cited

GenomicsPLoS GeneticsPeer-reviewedJul 6, 2020

Proteome-by-phenome Mendelian Randomisation detects 38 proteins with causal roles in human diseases and traits

Andrew D. Bretherick, Oriol Canela‐Xandri, Peter K. Joshi…·Chris Haley

45 citations

humanproteomics

GeneticsbioRxivPreprintJan 8, 2019

Genome-wide association analyses of risk tolerance and risky behaviors in over 1 million individuals identify hundreds of loci and shared genetic influences

Richard Karlsson Linnér, Pietro Biroli, Edward L. Kong…·Jonathan P. Beauchamp

GWAS in over one million individuals identify 611 loci across risk tolerance and risky behaviors, revealing shared genetic architecture and implicating glutamatergic and GABAergic neurotransmission.

82 citations

humanGWAS summary statistics

GenomicsbioRxivPreprintJul 6, 2018

Genomic underpinnings of lifespan allow prediction and reveal basis in modern risks

Paul R. H. J. Timmers, Ninon Mounier, Kristi Läll…·Peter K. Joshi

The paper identifies genetic loci associated with lifespan using genome-wide association and shows that variants linked to modern diseases explain most lifespan variation.

3 citations

humanGWAS summary statistics