Marta Barbara Wisniewska

The paper investigates TCF7L2 isoform redundancy in thalamic development, finding β-catenin-independent regulation and a developmental switch in activity.

This study investigates the role of TCF7L2 in thalamocortical energy metabolism and its impact on social behavior, finding that its loss leads to metabolic dysregulation and social deficits.

The paper identifies HAX1 RNA targets and shows its role in ribosome biogenesis and translation, linking it to disease mechanisms.

This study shows TCF7L2 regulates thalamic neuron differentiation and electrophysiological features by controlling gene expression and cell migration.