M. Shane Hutson

This study shows that cell fusion after wounding requires plasma membrane damage and endocytosis, with endocytosis expanding fusion pores to enable tissue repair.

The paper quantifies steroid hormone interactions with PDMS and models how these interactions disrupt dynamic dosing protocols in microfluidic devices.

The paper develops a toxicokinetic model to measure and predict chemical-PDMS interactions in organ-on-chip devices, showing accurate dose prediction under continuous-flow conditions.