Author

Tilman Breiderhoff

German Center for Neurodegenerative DiseasesORCID 0000-0002-1676-7498Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 1 Organism:mouse· 1

Frequent Co-Authors

Amr Al‐ShebeliD· 1 shared Dimitra PapaioannouiD· 1 shared Dominik MülleriD· 1 shared Jan HalbritteriD· 1 shared Michael M. KaminskiiD· 1 shared Philip BufleriD· 1 shared Phoebe Mossmann· 1 shared Sarah WendlingeriD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperMolecular Biology

Amr Al‐ShebelORCID

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1 paper

Recent·Most cited

Kidney-specific Wdr72 deletion leads to incomplete distal renal tubular acidosis through impaired V-ATPase B1 subunit localization

Amr Al‐Shebel, Phoebe Mossmann, Sarah Wendlinger, Dimitra Papaioannou, Tilman Breiderhoff, Wenjun Jin, Philip Bufler, Jan Halbritter, Michael M. Kaminski, Dominik Müller, Verena Klämbt

This study shows that kidney-specific deletion of Wdr72 causes incomplete distal renal tubular acidosis by reducing V-ATPase B1 subunit localization and impairing intercalated cell function.

Genes & Diseases · Jun 1, 2026Peer-reviewedMolecular Biology