Author

Simon J. Tunster

University of CambridgeORCID 0000-0002-2242-9452Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1

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Amanda Nancy Sferruzzi-PerriiD· 1 shared Bethany R. L. Aykroyd· 1 shared

Activity by Year

2022-2022 · peak 1/yr

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1 shared paperDevelopmental Biology

Amanda Nancy Sferruzzi-PerriORCID

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1 paper

Recent·Most cited

Loss of imprinting of the Igf2-H19 ICR1 enhances placental endocrine capacity via sex-specific alterations in signalling pathways in the mouse.

Bethany R. L. Aykroyd, Simon J. Tunster, Amanda Nancy Sferruzzi-Perri

This study shows that loss of imprinting at the Igf2-H19 ICR1 in mouse placental endocrine junctional zone leads to sex-specific changes in signaling pathways affecting endocrine capacity.

Development · Jan 1, 2022Peer-reviewedDevelopmental Biology