Author

Pascual Sanz

Consejo Superior de Investigaciones CientíficasORCID 0000-0002-2399-4103Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1

Frequent Co-Authors

Ángela Campos‐Rodríguez· 1 shared Carmina Franch-IbáñeziD· 1 shared Helena MiraiD· 1 shared Isabel Calatayud-BaselgaiD· 1 shared José Guijarro-NueziD· 1 shared Lucía Casares-CrespoiD· 1 shared Rosa VianaiD· 1 shared Teresa RubioiD· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

8 top collaborators · 13 mutual co-author links

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1 shared paperNeuroscience

Ángela Campos‐Rodríguez

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2 papers

Recent·Most cited

Glial alterations in the glutamatergic and GABAergic signaling pathways in a mouse model of Lafora disease, a severe form of progressive myoclonus epilepsy

Rosa Viana, Teresa Rubio, Ángela Campos‐Rodríguez, Pascual Sanz

The paper investigates glial alterations in glutamatergic and GABAergic signaling pathways in a mouse model of Lafora disease, finding dysregulated receptor subunits and increased GABA transporter levels in glial cells.

bioRxiv · Sep 13, 2024 · PreprintNeuroscience

Autophagy is a cell-intrinsic driver of neural stem cell quiescence in hippocampal dentate gyrus development

Isabel Calatayud-Baselga, Lucía Casares-Crespo, Carmina Franch-Ibáñez, José Guijarro-Nuez, Pascual Sanz, Helena Mira

This study shows autophagy is essential for establishing quiescence in hippocampal neural stem cells during development.

bioRxiv · May 3, 2023 · PreprintNeuroscience