Author

Marwa Elmasri

University of SussexORCID 0000-0002-2582-7640Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Marwa Elmasri1 paper · UCL Queen Square Institute of Neurology

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Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 1 Assay:electronic health records· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

Andrew C. PenniD· 1 shared Daniel HunteriD· 1 shared Does Moolenaar Van Der Does· 1 shared Eirini Karachaliou· 1 shared Ella Emine Bates· 1 shared Giles WinchesteriD· 1 shared Kenji SakimuraiD· 1 shared Wajeeha AziziD· 1 shared

Activity by Year

2022-2022 · peak 1/yr

Co-Author Collaboration Network

8 top collaborators · 28 mutual co-author links

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1 shared paperNeuroscience

Andrew C. PennORCID

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1 paper

Recent·Most cited

Common synaptic phenotypes arising from diverse mutations in the human NMDA receptor subunit GluN2A

Marwa Elmasri, Daniel Hunter, Giles Winchester, Ella Emine Bates, Wajeeha Aziz, Does Moolenaar Van Der Does, Eirini Karachaliou, Kenji Sakimura, Andrew C. Penn

The paper examines how different mutations in the GluN2A subunit of the NMDA receptor affect synaptic currents in hippocampal neurons, finding prolonged current decays as a common effect.

Communications Biology · Feb 28, 2022Peer-reviewedNeuroscience