Author

Ellen Heilmann Modalsli

Norwegian University of Science and TechnologyORCID 0000-0002-3061-1844Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetic & Genomic Medicine· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Research Focus:GWAS / association mapping· 2 Assay:GWAS summary statistics· 2 Organism:human· 2 Approach:statistical model· 2

Frequent Co-Authors

James T. ElderiD· 2 shared Lam C. TsoiiD· 2 shared Mari LøsetiD· 2 shared Sara BrowniD· 2 shared Stefan SiebertiD· 2 shared Allison C. BilliiD· 1 shared André FrankeiD· 1 shared André ReisiD· 1 shared

Activity by Year

2018-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersGenetic & Genomic Medicine

James T. ElderORCID

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2 papers

Recent·Most cited

Genetic & Genomic MedicineNature CommunicationsPeer-reviewedFeb 28, 2025

GWAS meta-analysis of psoriasis identifies new susceptibility alleles impacting disease mechanisms and therapeutic targets

Nick Dand, Philip E. Stuart, John Bowes…·James T. Elder

This study performs a GWAS meta-analysis of psoriasis, identifying 109 susceptibility loci, including 45 new ones, and highlights potential therapeutic targets and disease mechanisms.

107 citationstop 1%

humanGWAS summary statistics

𝕏 PostBluesky

GeneticsbioRxivPreprintFeb 15, 2018

Evidence of a common causal relationship between body mass index and inflammatory skin disease: a Mendelian Randomization study

Ashley Budu‐Aggrey, Ben Michael Brumpton, Jessica Tyrrell…·Lavinia Paternoster

This study uses Mendelian Randomization to show that higher BMI causally increases the risk of psoriasis and eczema, with a stronger effect on psoriasis.

5 citations

humanGWAS summary statistics

𝕏 PostBluesky