Author

Fay G. Newton

Institute of Genetics and CancerORCID 0000-0002-3295-8657Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Research Focus:cell-cell communication· 1 Organism:Drosophila· 1 Approach:experimental assay· 1 Organism:mouse· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Alyona KederiD· 1 shared Anastasia FiliaiD· 1 shared Andrew P. JarmaniD· 1 shared Assel Kashkenbayeva· 1 shared Camille H. Tardieu· 1 shared Joerg T. AlbertiD· 1 shared Jonathan E. GaleiD· 1 shared Linda NguyeniD· 1 shared

Activity by Year

2020-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperNeuroscience

Alyona KederORCID

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2 papers

Recent·Most cited

Autophagy disruption and mitochondrial stress precede photoreceptor necroptosis in multiple mouse models of inherited retinal disorders.

Fay G. Newton, Mihail R. Halachev, Linda Nguyen, Lisa McKie, Pleasantine Mill, Roly D. Megaw

The paper identifies common early defects in autophagy and mitochondrial damage in multiple IRD models, leading to necroptosis and photoreceptor loss, suggesting potential gene-agnostic therapeutic targets.

Nature Communications · Apr 29, 2025Peer-reviewedCell Biology

Homeostatic maintenance and age-related functional decline in the Drosophila ear

Alyona Keder, Camille H. Tardieu, Liza Malong, Anastasia Filia, Assel Kashkenbayeva, Fay G. Newton, Marcos Georgiades, Jonathan E. Gale, Michael Lovett, Andrew P. Jarman, Joerg T. Albert

This study identifies key transcription factors involved in maintaining auditory function in Drosophila and shows their role in age-related hearing loss.

Scientific Reports · May 4, 2020Peer-reviewedNeuroscience