Author

Young‐Woo Jo

Seoul National UniversityORCID 0000-0002-4230-8682Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Sang‐Hyeon HanniD· 2 shared Ye Lynne KimiD· 2 shared Young‐Yun KongiD· 2 shared Daehyun Baek· 1 shared Hye‐Rim ParkiD· 1 shared In-Wook Song· 1 shared Inkuk ParkiD· 1 shared Ji-Hoon KimiD· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersNeuroscience

Sang‐Hyeon HannORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Muscle-resident mesenchymal progenitors sense and repair peripheral nerve injury via the GDNF-BDNF axis

Kyusang Yoo, Young‐Woo Jo, Takwon Yoo, Sang‐Hyeon Hann, Inkuk Park, Yea-Eun Kim, Ye Lynne Kim, Joonwoo Rhee, In-Wook Song, Ji-Hoon Kim, Daehyun Baek, Young‐Yun Kong

This study identifies a subset of muscle-resident FAPs that respond to peripheral nerve injury via the GDNF-BDNF axis, contributing to remyelination and nerve regeneration.

eLife · Aug 30, 2024Peer-reviewedNeuroscience

Depletion of SMN Protein in Mesenchymal Progenitors Impairs the Development of Bone and Neuromuscular Junction in Spinal Muscular Atrophy

Sang‐Hyeon Hann, Seon-Yong Kim, Ye Lynne Kim, Young‐Woo Jo, Jong‐Seol Kang, Hye‐Rim Park, Se‐Young Choi, Young‐Yun Kong

This study shows that SMN deficiency in mesenchymal progenitors causes bone and neuromuscular junction defects in SMA, with potential therapeutic implications.

eLife · Nov 7, 2023Peer-reviewedNeuroscience