Author

Young‐Yun Kong

Seoul National UniversityORCID 0000-0001-7335-3729Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 3 Developmental Biology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 4 Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Assay:spatial transcriptomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Kyusang YooiD· 3 shared Sang‐Hyeon HanniD· 3 shared Ye Lynne KimiD· 3 shared Daehyun Baek· 2 shared In-Wook Song· 2 shared Se‐Young ChoiiD· 2 shared Seon-Yong KimiD· 2 shared Takwon YooiD· 2 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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3 shared papersNeuroscience

Kyusang YooORCID

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4 papers

Recent·Most cited

NeurosciencebioRxivPreprintJun 19, 2026

Terminal Schwann Cells Regulate Presynaptic Vesicle Homeostasis but Not Neuromuscular Junction Integrity in Mice

Hyun Kim, Seon-Yong Kim, Kyusang Yoo…·Young‐Yun Kong

This study identifies Col20a1 as a marker for terminal Schwann cells and shows they regulate presynaptic vesicle homeostasis but not neuromuscular junction integrity in mice.

DOI 10.64898/2026.06.15.732329

mousespatial transcriptomics

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Developmental BiologybioRxivPreprintAug 29, 2025

Rejuvenation-Responsive and Senolytic-Sensitive Muscle Stem Cells Unveiled by CD200 and CD63 in Geriatric Muscle

Ye Lynne Kim, Young-Woo Jo, Takwon Yoo…·Young‐Yun Kong

The paper identifies a new subpopulation of aged muscle stem cells (GERI-MuSCs) that are senolytic-sensitive and rejuvenation-responsive, characterized by CD200 and CD63 markers.

DOI 10.1101/2025.08.29.673011

mousebulk RNA-seq

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NeuroscienceeLifePeer-reviewedAug 30, 2024

Muscle-resident mesenchymal progenitors sense and repair peripheral nerve injury via the GDNF-BDNF axis

Kyusang Yoo, Young‐Woo Jo, Takwon Yoo…·Young‐Yun Kong

This study identifies a subset of muscle-resident FAPs that respond to peripheral nerve injury via the GDNF-BDNF axis, contributing to remyelination and nerve regeneration.

10 citations

mousebulk RNA-seq

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NeuroscienceeLifePeer-reviewedNov 7, 2023

Depletion of SMN Protein in Mesenchymal Progenitors Impairs the Development of Bone and Neuromuscular Junction in Spinal Muscular Atrophy

Sang‐Hyeon Hann, Seon-Yong Kim, Ye Lynne Kim…·Young‐Yun Kong

This study shows that SMN deficiency in mesenchymal progenitors causes bone and neuromuscular junction defects in SMA, with potential therapeutic implications.

5 citations

mouseexperimental assay

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