Author

Huaye Zhang

Rutgers, The State University of New JerseyORCID 0000-0002-4844-6111Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:spatial domain detection· 1 Approach:statistical model· 1

Frequent Co-Authors

Zhiping P. PangiD· 2 shared Ankitha H. DindigaliD· 1 shared Elif Naz OzseniD· 1 shared Emily C. Kelly-CastroiD· 1 shared George C. WagneriD· 1 shared Hanna CaiolaiD· 1 shared Junlong LiiD· 1 shared Kevin MonahaniD· 1 shared

Activity by Year

2024-2024 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersNeuroscience

Zhiping P. PangORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Neuronal connectivity, behavioral, and transcriptional alterations associated with the loss of MARK2

Hanna Caiola, Qian Wu, Junlong Li, Xuefeng Wang, Shaili Soni, Kevin Monahan, George C. Wagner, Zhiping P. Pang, Huaye Zhang

This study shows that loss of MARK2 in vivo causes dendritic spine changes, reduced excitatory transmission, and behavioral impairments, including seizures and cognitive deficits.

The FASEB Journal · Oct 22, 2024Peer-reviewedNeuroscience

Loss of the polarity protein Par3 promotes dendritic spine neoteny and enhances learning and memory

Mikayla M. Voglewede, Elif Naz Ozsen, Noah A. Ivak, Matteo Bernabucci, Ruizhe Tang, Miao Sun, Zhiping P. Pang, Huaye Zhang

This study shows that loss of Par3 increases long, thin dendritic spines and enhances hippocampal-dependent learning and memory in vivo.

iScience · Jun 19, 2024Peer-reviewedNeuroscience

MARK1 regulates dendritic spine morphogenesis and cognitive functions in vivo

Emily C. Kelly-Castro, Rebecca Shear, Ankitha H. Dindigal, Maitreyee Bhagwat, Huaye Zhang

This study shows that MARK1 is essential for dendritic spine morphogenesis and cognitive functions in vivo, as evidenced by spine defects and behavioral impairments in Mark1 knockout mice.

Experimental Neurology · Mar 12, 2024Peer-reviewedNeuroscience