Author

Paola Cappello

University of TurinORCID 0000-0002-5321-7794Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Research Focus:clustering· 1 Approach:deep learning· 1

Frequent Co-Authors

Alicia TayiD· 1 shared Antanas GulbinasiD· 1 shared Basil Kuok Zi XiiD· 1 shared Chong Xiang TaniD· 1 shared Denis M. GrantiD· 1 shared Elisavet Kalaitsidou· 1 shared Fauzia Nurul IzzatiiD· 1 shared Germaine Teng· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCancer Biology

Alicia TayORCID

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintMar 9, 2026

Multi-omics and functional analysis of a bioengineered vascularized pancreatic cancer model reveal an immunosuppressive and therapy-resistant niche

Giulio Giustarini, Kok Siong Ang, Pavanish Kumar…·Giulia Adriani

The paper develops a bioengineered vascularized pancreatic cancer model and uses multi-omics and functional assays to reveal an immunosuppressive and therapy-resistant tumor niche.

1 citation

humanscRNA-seq

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Cancer BiologyFrontiers in ImmunologyPeer-reviewedJan 10, 2025

Loss of PARP7 increases type I interferon signalling and prevents pancreatic tumour growth by enhancing immune cell infiltration

Vinicius Kannen, Marit Rasmussen, Siddhartha Das…·Jason B. Matthews

This study shows that loss of PARP7 in pancreatic cancer cells increases type I interferon signaling and enhances immune cell infiltration, leading to reduced tumor growth.

5 citations

mousebulk RNA-seq

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