Author

Jun Suzuki

Kyoto UniversityORCID 0000-0002-5441-9486Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:Drosophila· 1 Organism:mouse· 1

Frequent Co-Authors

Akimitsu Tsumadori· 1 shared Akinao NoseiD· 1 shared Etsu Matsutani· 1 shared Ghazaleh BehnammaneshiD· 1 shared Hiroshi KohsakaiD· 1 shared Jonathan E. BirdiD· 1 shared Kaho MaetaiD· 1 shared Kai LiiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperNeuroscience

Akimitsu Tsumadori

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2 papers

Recent·Most cited

KDM6A Haploinsufficiency Is Insufficient to Disrupt Cochlear Structure and Auditory Function in Mouse Models of Kabuki Syndrome Type 2

Yuichi Shimizu, Shun Wakamori, Loryn Smith, Ghazaleh Behnammanesh, Yohei Honkura, Jun Suzuki, Jonathan E. Bird, Yukio Katori, Shinichi Someya

This study examines whether Kdm6a deficiency causes cochlear pathology or auditory dysfunction in mouse models of Kabuki syndrome type 2, finding no overt cochlear or auditory issues in viable models.

bioRxiv · Oct 12, 2026 · PreprintDevelopmental Biology

The membrane-tethered cis-ligand Belly roll elicits GPCR signaling thereby enabling adaptive avoidance behaviors

Yuma Tsukasa, Takumi Ohbayashi, Misato Kurio, Hiroshi Kohsaka, Kaho Maeta, Etsu Matsutani, Akimitsu Tsumadori, Kai Li, Risa Matsui, Jun Suzuki, Akinao Nose, Tadashi Uemura, Tadao Usui

The paper identifies a neural circuit and molecular mechanism in Drosophila larvae that links internal osmotic state to behavioral adaptation through GPCR signaling.

bioRxiv · Jul 3, 2026 · PreprintNeuroscience