Akinori Kawamura

This study shows that extensive disruption of HoxA/D genes in zebrafish does not cause visible defects in anterior vertebral patterning, suggesting HoxB/C genes play a more critical role.

This study shows that hoxba and hoxbb clusters are essential for positioning zebrafish pectoral fins by inducing tbx5a expression and maintaining retinoic acid responsiveness.

The study identifies a transposon insertion in the hoxc12a 3' UTR of widefins medaka that reduces hox gene expression and causes phenotypic variation in fin rays.

This study identifies RA-responsive Hox genes in zebrafish that are essential for pharyngeal pouch formation and reveal their regulatory relationship with pax1.

This study shows that the size and position of the zebrafish swim bladder are physically constrained by surrounding bones during development.

This study investigates the role of HoxA- and HoxD-related clusters in zebrafish pectoral fin development by analyzing mutants with combined gene deletions and finds that their simultaneous deletion causes significant developmental defects.