Robert M. Blanton

This study identifies T cell-derived IFNγ as a driver of ECM stiffening in HFpEF via LOXL3 in cardiac fibroblasts, showing therapeutic potential for targeting this pathway.

This study identifies cGMP reduction as a driver of VT in DMII and metabolic syndrome, showing that pharmacological modulation of cGMP-PKG1alpha pathways can reduce VT inducibility.

This study uses PET-MRI to detect cardiopulmonary fibrosis in a mouse model of left ventricular dysfunction, showing increased signals correlate with fibrotic markers.