Author

Ulrika Nordström

Umeå UniversityORCID 0000-0002-7757-2344Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:protein structure· 1 Research Focus:protein structure prediction· 1 Assay:proteomics· 1

Frequent Co-Authors

Per ZetterströmiD· 2 shared Peter Munch AnderseniD· 2 shared Stefan L. MarklundiD· 2 shared Agneta Öberg· 1 shared Dale J. LangeiD· 1 shared Elin Forsgren· 1 shared Eva Jonsson· 1 shared Isil KeskiniD· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersNeuroscience

Per ZetterströmORCID

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2 papers

Recent·Most cited

SOD1 Enzymatic Activity in CSF from ALS patients with and without SOD1 mutations

Laura Leykam, Karin Forsberg, Ulrika Nordström, Karin Hjertkvist, Agneta Öberg, Eva Jonsson, Peter Munch Andersen, Stefan L. Marklund, Per Zetterström

The paper presents a method to measure SOD1 enzymatic activity in CSF from ALS patients, showing lower activity in mutant SOD1 carriers despite normal protein levels.

Neurobiology of Disease · Oct 28, 2024Peer-reviewedNeuroscience

The molecular pathogenesis of superoxide dismutase 1-linked ALS is promoted by low oxygen tension

Isil Keskin, Elin Forsgren, Manuela Lehmann, Peter Munch Andersen, Thomas Brännström, Dale J. Lange, Matthis Synofzik, Ulrika Nordström, Per Zetterström, Stefan L. Marklund, Jonathan D. Gilthorpe

This study shows that low oxygen tension promotes SOD1 misfolding and aggregation in ALS-related cells, linking oxygen levels to disease pathogenesis.

Acta Neuropathologica · Mar 12, 2019Peer-reviewedNeuroscience