NeuroscienceJournal of NeurosciencePeer-reviewed
Deletion of the mitochondrial matrix protein cyclophilin-D prevents parvalbumin interneuron dysfunction and cognitive deficits in a mouse model of NMDA hypofunction
Aarron J. Phensy, Kathy L. Lindquist, Karen A. Lindquist…·Sven Kroener
This study shows that deleting cyclophilin-D in mice prevents cognitive and synaptic deficits caused by perinatal NMDA receptor blockade.
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