Author

Kathy L. Lindquist

The University of Texas at DallasORCID 0000-0002-1213-6664Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Organism:rat· 1

Frequent Co-Authors

Aarron J. PhensyiD· 2 shared Sven KroeneriD· 2 shared Aurian O. NaderiiD· 1 shared Bogdan BordieanuiD· 1 shared Christopher M. DriskilliD· 1 shared Dania BairutyiD· 1 shared Esha GaubaiD· 1 shared Heng DuiD· 1 shared

Activity by Year

2020-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersNeuroscience

Aarron J. PhensyORCID

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3 papers

Recent·Most cited

NeurosciencebioRxivPreprintNov 7, 2025

Rescuing cognitive deficits in alcohol-dependent mice by targeting physiological adaptations in prefrontal cortex KV7 channels

Kathy L. Lindquist, Sudarat Nimitvilai, Hyland C. Gonzalez…·Patrick J. Mulholland

This study shows that targeting KV7 channels in the prefrontal cortex of alcohol-dependent mice can rescue cognitive deficits and reduce alcohol intake.

DOI 10.1101/2025.11.07.687180

mouseexperimental assay

𝕏 PostBluesky

NeuroscienceJournal of NeurosciencePeer-reviewedMay 7, 2024

Vagus nerve stimulation (VNS) modulates synaptic plasticity in the rat infralimbic cortex via Trk-B receptor activation to reduce drug-seeking

Christopher M. Driskill, Jessica E. Childs, Aarron J. Phensy…·Sven Kroener

This study shows that vagus nerve stimulation (VNS) enhances extinction learning and reduces drug-seeking in rats by increasing BDNF and modulating TrkB receptors in the infralimbic cortex.

8 citations

ratexperimental assay

𝕏 PostBluesky

NeuroscienceJournal of NeurosciencePeer-reviewedJun 30, 2020

Deletion of the mitochondrial matrix protein cyclophilin-D prevents parvalbumin interneuron dysfunction and cognitive deficits in a mouse model of NMDA hypofunction

Aarron J. Phensy, Kathy L. Lindquist, Karen A. Lindquist…·Sven Kroener

This study shows that deleting cyclophilin-D in mice prevents cognitive and synaptic deficits caused by perinatal NMDA receptor blockade.

13 citations

mouseexperimental assay

𝕏 PostBluesky