Author

Kai Fu

University of California, Los AngelesORCID 0000-0002-8107-3556Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Kai Fu1 paper · Yunnan University

Biology Disciplines

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Developmental Biology· 1 Genomics· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Assay:ChIP-seq· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Constantinos ChronisiD· 2 shared Kathrin PlathiD· 2 shared Matteo A. PellegriniiD· 2 shared Abdenour SoufiiD· 1 shared Benni S. Vargas· 1 shared Carlos Salas· 1 shared Denise E. AlleniD· 1 shared Edward KuoyiD· 1 shared

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2017-2018 · peak 1/yr

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10 top collaborators · 35 mutual co-author links

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2 shared papersDevelopmental Biology

Constantinos ChronisORCID

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2 papers

Recent·Most cited

Developmental BiologyStem Cell ReportsPeer-reviewedMay 1, 2018

Loss of MECP2 leads to induction of p53 and cell senescence

Minori Ohashi, Elena Korsakova, Denise E. Allen…·William E. Lowry

This study shows that loss of MECP2 in neurons leads to p53 induction and cell senescence, causing dendritic defects.

80 citationstop 4%

humanbulk RNA-seq

GenomicsbioRxivPreprintNov 27, 2017

Comparison of reprogramming factor targets reveals both species-specific and conserved mechanisms in early iPS cells

Kai Fu, Constantinos Chronis, Abdenour Soufi…·Matteo A. Pellegrini

This study compares OSKM reprogramming factor binding in human and mouse fibroblasts, finding conserved mechanisms and species-specific differences in target sites and regulatory networks.

DOI 10.1101/225326