Author

Rachael E. Redgrave

Centre for LifeORCID 0000-0002-8539-4971Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:differential expression· 1 Assay:proteomics· 1

Frequent Co-Authors

Helen M. PhillipsiD· 2 shared Andrew D.H. Morgan· 1 shared Dominique RolandiD· 1 shared Esha Singh· 1 shared Gavin D. RichardsoniD· 1 shared George MercesiD· 1 shared Helen M. ArthuriD· 1 shared John BurniD· 1 shared

Activity by Year

2020-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersGenetics

Helen M. PhillipsORCID

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2 papers

Recent·Most cited

A Slc5a6 Deficient Mouse Model Reveals a Metabolically Driven Dilated Cardiomyopathy with Therapeutic Potential for Vitamin-Based Intervention

Millie Fullerton, Lauren C. Phillips, Rachael E. Redgrave, Luke Spray, Vincent Haufroid, George Merces, Scott T. Kerridge, Gavin D. Richardson, Nathalie Mercier, Dominique Roland, Rebecca Crossley, Andrew D.H. Morgan, Joseph P. Dewulf, John Burn, Simon D. Bamforth, Helen M. Phillips

This study shows that SLC5A6 deficiency causes dilated cardiomyopathy in mice, which can be prevented by vitamin supplementation, mirroring clinical outcomes in patients.

JCI Insight · Jun 2, 2026Peer-reviewedGenetics

Arterial endoglin does not protect against arteriovenous malformations

Esha Singh, Rachael E. Redgrave, Helen M. Phillips, Helen M. Arthur

The paper shows that arterial endoglin (ENG) is not necessary to prevent arteriovenous malformations (AVMs), as specific loss of ENG in venous and capillary endothelium resulted in AVM frequency similar to pan-endothelial loss.

Angiogenesis · Jun 6, 2020Peer-reviewedGenetics