Author

Mohamed Hammad

City Of Hope National Medical CenterORCID 0000-0002-9684-2506Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Approach:mechanistic / dynamical model· 1

Frequent Co-Authors

Eslam E. Abd El-FattahiD· 2 shared Chia‐en A. ChangiD· 1 shared Karen Saida AboodyiD· 1 shared Kingsley Wu· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

4 top collaborators · 6 mutual co-author links

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2 shared papersCancer Biology

Eslam E. Abd El-FattahORCID

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintApr 22, 2026

CACNA1C as a Prognostic Biomarker and Therapeutic Target in High-Grade Serous Ovarian Cancer: Clinical Validation and Molecular Dynamics of Nifedipine Blockade

Mohamed Hammad, Kingsley Wu, Eslam E. Abd El-Fattah…·Chia‐en A. Chang

This paper identifies CACNA1C as a prognostic biomarker and therapeutic target in high-grade serous ovarian cancer, showing nifedipine suppresses tumor growth through calcium channel blockade.

DOI 10.64898/2026.04.19.719516

humanbulk RNA-seq

Cancer BiologybioRxivPreprintMar 31, 2026

Tumor-Intrinsic IL-17 Signaling Correlates with Multimodal Resistance Phenotypes Following Oncolytic Adenovirus Challenge

Eslam E. Abd El-Fattah, Mohamed Hammad

This study identifies IL-17 signaling as a tumor-intrinsic factor associated with resistance to oncolytic adenovirus therapy, linked to stemness, metabolic changes, and reduced cell death.

DOI 10.64898/2026.03.27.714871

mousebulk RNA-seq