Author

Iván Quesada

Instituto de Salud Carlos IIIORCID 0000-0002-9808-514XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Ángel NadaliD· 1 shared Daniel Martínez-Bañón· 1 shared Elena Ribes-García· 1 shared Hilda FerreroiD· 1 shared Juan Martínez‐PinnaiD· 1 shared Mónica Serrano-Selva· 1 shared Paloma Alonso‐MagdalenaiD· 1 shared R Betoret Gustems· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperPhysiology

Ángel NadalORCID

Mutual co-authors in cluster

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1 paper

Recent·Most cited

Increased TGFβ /Activin-Smad2 signaling is associated with pancreatic β-cell dysfunction and glucose intolerance in gestational diabetes mellitus

Talía Boronat‐Belda, Hilda Ferrero, Sergi Soriano, Elena Ribes-García, R Betoret Gustems, Daniel Martínez-Bañón, Mónica Serrano-Selva, Juan Martínez‐Pinna, Ángel Nadal, Iván Quesada, Paloma Alonso‐Magdalena

This study identifies increased TGFβ/Activin-Smad2 signaling in GDM as a contributor to β-cell dysfunction and glucose intolerance, with pharmacological inhibition showing therapeutic potential.

Molecular Metabolism · Oct 18, 2025Peer-reviewedPhysiology