Author

Paloma Alonso‐Magdalena

Instituto de Salud Carlos IIIORCID 0000-0003-1065-5388Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1

Frequent Co-Authors

Talía Boronat‐BeldaiD· 2 shared Ana María Sartorius-Yáñez· 1 shared Ángel NadaliD· 1 shared Beatriz MerinoiD· 1 shared Christophe BrocaiD· 1 shared Daniel Martínez-Bañón· 1 shared Elena Ribes-García· 1 shared Germán PerdomoiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersPhysiology

Talía Boronat‐BeldaORCID

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2 papers

Recent·Most cited

PhysiologybioRxivPreprintSep 15, 2026

Preimplantation factor (PIF) links embryo-derived signaling to maternal pancreatic β-cell adaptation through an ERα-dependent pathway

Raquel Pascua‐Maestro, Ana María Sartorius-Yáñez, Silvia Hernández de la Red…·Beatriz Merino

This study identifies PIF as an embryo-derived signal that regulates maternal β-cell adaptation during pregnancy, showing its role in β-cell proliferation and insulin secretion through an ERα-dependent pathway.

DOI 10.64898/2026.09.10.750557

mouseexperimental assay

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PhysiologyMolecular MetabolismPeer-reviewedOct 18, 2025

Increased TGFβ /Activin-Smad2 signaling is associated with pancreatic β-cell dysfunction and glucose intolerance in gestational diabetes mellitus

Talía Boronat‐Belda, Hilda Ferrero, Sergi Soriano…·Paloma Alonso‐Magdalena

This study identifies increased TGFβ/Activin-Smad2 signaling in GDM as a contributor to β-cell dysfunction and glucose intolerance, with pharmacological inhibition showing therapeutic potential.

DOI 10.1016/j.molmet.2025.102274

mousehuman

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