Molly M. McGinnis

The paper examines how chronic ethanol exposure affects synaptic plasticity in the AIC-BLA circuit during withdrawal and shows that ketamine prevents these changes and associated anxiety-like behavior.

This study shows chronic ethanol exposure and withdrawal increase glutamate release from dorsal mPFC terminals but decrease it from ventral mPFC terminals in the BLA, affecting anxiety-like behavior.

This study shows chronic ethanol exposure enhances glutamate release from thalamic inputs to the rat lateral amygdala, and manipulating these inputs reduces withdrawal-related anxiety.