Timothy D. O’Connell

This study shows that Ffar4 in cardiac myocytes reduces systolic dysfunction after ischemia-reperfusion by improving mitochondrial function and cGMP signaling.

This study shows that Ffar4 and ChemR23 signaling in cardiac myocytes protects against ischemic injury by reducing oxidative stress and cell death.

The paper identifies Endoglin as an inhibitor of cardiomyocyte proliferation and shows its role in cardiac regeneration through genetic and mechanistic studies.