Author

Gunnhild Åberge Vie

Norwegian University of Science and TechnologyORCID 0000-0003-1552-5291Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetics· 2

Signature Assays, Organisms & Methods

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Assay:GWAS summary statistics· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:benchmarking· 1 Research Focus:GWAS / association mapping· 1

Frequent Co-Authors

Ben Michael BrumptoniD· 2 shared Bjørn Olav ÅsvoldiD· 2 shared George Davey SmithiD· 2 shared Adam AutoniD· 1 shared Alexandra HavdahliD· 1 shared Amanda HughesiD· 1 shared Andrew David GrotzingeriD· 1 shared Andrew R. WoodiD· 1 shared

Activity by Year

2018-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersGenetics

Ben Michael BrumptonORCID

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2 papers

Recent·Most cited

GeneticsNature CommunicationsPeer-reviewedJul 14, 2020

Within-family studies for Mendelian randomization: avoiding dynastic, assortative mating, and population stratification biases

Ben Michael Brumpton, Eleanor C. M. Sanderson, Karl Heilbron…·Neil M Davies

This paper introduces within-family Mendelian randomization methods to reduce biases from family structure and shows they yield different results compared to population-based analyses.

402 citationstop 1%

humanGWAS summary statistics

𝕏 PostBluesky

GeneticsbioRxivPreprintFeb 15, 2018

Evidence of a common causal relationship between body mass index and inflammatory skin disease: a Mendelian Randomization study

Ashley Budu‐Aggrey, Ben Michael Brumpton, Jessica Tyrrell…·Lavinia Paternoster

This study uses Mendelian Randomization to show that higher BMI causally increases the risk of psoriasis and eczema, with a stronger effect on psoriasis.

5 citations

humanGWAS summary statistics

𝕏 PostBluesky