Author

Ricardo Blázquez‐Encinas

Instituto Maimónides de Investigación Biomédica de CórdobaORCID 0000-0003-2904-8625Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Research Focus:differential expression· 3 Research Focus:gene regulatory network inference· 3 Organism:human· 3 Approach:statistical model· 3 Research Focus:cell type annotation· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:clustering· 1

Frequent Co-Authors

Alejandro Ibáñez‐CostaiD· 3 shared Justo Pastor CastanoiD· 3 shared Sergio Pedraza‐ArévaloiD· 3 shared Víctor García‐VioqueiD· 3 shared Aldo ScarpaiD· 2 shared María Trinidad Moreno-MontillaiD· 2 shared Rita Teresa LawloriD· 2 shared Alejandro García‐ÁlvareziD· 1 shared

Activity by Year

2024-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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3 shared papersCancer Biology

Alejandro Ibáñez‐CostaORCID

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3 papers

Recent·Most cited

Cancer BiologybioRxivPreprintOct 25, 2026

From Splicing to Survival: Understanding the Molecular Basis of Prognostic Differences in advanced Small Intestine Neuroendocrine Tumors

Víctor García‐Vioque, Alejandro García‐Álvarez, Maria Trinidad Moreno-Montilla…·Justo Pastor Castano

This study investigates the relationship between spliceosomic changes and patient survival in advanced SI-NETs, identifying CPSF2 as a key factor linked to poor prognosis.

DOI 10.64898/2026.10.22.760955

humanbulk RNA-seq

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Cancer BiologyEndocrine PathologyPeer-reviewedJul 9, 2026

An integrative RNA spliceosomic landscape of pancreatic neuroendocrine tumors unveils novel clinicomolecular associations

Ricardo Blázquez‐Encinas, Víctor García‐Vioque, Andrea Mafficini…·Justo Pastor Castano

This study identifies three distinct spliceosomic profiles in pancreatic neuroendocrine tumors linked to clinical and molecular characteristics and potential therapeutic targets.

1 citation

humanbulk RNA-seq

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Cancer BiologyCancer Gene TherapyPeer-reviewedSep 4, 2024

The Exon Junction Complex component EIF4A3 plays a splicing-linked oncogenic role in Pancreatic Ductal Adenocarcinoma

Ricardo Blázquez‐Encinas, Emilia Alors‐Pérez, María Trinidad Moreno-Montilla…·Justo Pastor Castano

This study identifies EIF4A3 as an oncogenic factor in PDAC by showing its overexpression correlates with poor prognosis and its role in regulating RNA splicing and tumor growth.

9 citations

humanbulk RNA-seq

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