Brendan D. Adkinson

This study shows ketamine broadens neural spatial tuning and impairs spatial working memory in healthy humans, linking NMDAR antagonism to cortical microcircuit disruptions.

The paper identifies inflated effect sizes in functional neuroimaging studies and proposes a correction method to improve study planning and detection of brain effects.

This study analyzes self-citation rates across 100,347 articles from 63 neuroscience journals between 2000-2020, revealing trends in self-citation by author position, country, gender, and field.

The paper shows that overlooked brain connectivity features can yield different neurobiological interpretations with similar prediction accuracy in neuroimaging studies.

The paper evaluates the statistical power of external validation in brain-phenotype predictions and finds that sample size impacts power and effect size inflation.

This study investigates how ketamine induces distinct whole-brain functional connectivity patterns in individuals and links these patterns to gene expression and behavioral responses.

The paper tests predictive models' ability to generalize across diverse, unharmonized neuroimaging datasets and finds that cross-dataset training improves prediction robustness.

This paper improves brain-behavior prediction by imputing missing data in connectome-based models, increasing sample size and explained variance.

CAROT remaps connectomes between atlases without raw data, enabling reuse of processed data across different atlases.

The paper develops a stable brain-behavior space mapping for psychosis-spectrum disorders using symptom and brain data, showing univariate approaches outperform multivariate ones for individualized prediction.

The paper develops a dynamical circuit model of human cortex incorporating hierarchical heterogeneity to better explain large-scale neural dynamics and functional connectivity.

This study shows that LSD-induced changes in brain connectivity are mediated by the 5-HT2A receptor, as demonstrated by the blocking effect of a 5-HT2A antagonist.