Karine Fénelon

This study identifies PPTg glutamatergic neurons as critical modulators of PPI in mice, showing their role in shaping startle responses through time-dependent effects on PnC neurons.

The paper identifies CeA-PnC glutamatergic synapses as a therapeutic target for reversing PPI deficits in schizophrenia models using photo-tagging and PRODH-deficient mice.

This study identifies brainstem glycinergic neurons critical for prepulse inhibition (PPI) in young mice, showing their reduced role in age-related PPI decline.

This study investigates the role of amygdalar excitatory projections to the PnC in PPI, finding that optogenetic manipulation of these connections affects PPI behavior.