Author

Meleah A. Hickman

Emory UniversityORCID 0000-0003-4237-6981Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:C. elegans· 2 Research Focus:variant calling· 2 Assay:whole-genome sequencing· 2 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Approach:statistical model· 1

Frequent Co-Authors

Amanda C SmithiD· 1 shared Dorian J. FeisteliD· 1 shared Levi T. MorraniD· 1 shared Ognenka AvramovskaiD· 1 shared Rema Elmostafa· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

5 top collaborators · 2 mutual co-author links

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1 shared paperGenomics

Amanda C SmithORCID

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3 papers

Recent·Most cited

Host defense mechanisms induce genome instability in an opportunistic fungal pathogen

Amanda C Smith, Levi T. Morran, Meleah A. Hickman

This study shows that host-produced reactive oxygen species and antimicrobial peptides induce genome instability in Candida albicans, leading to genetic changes that may enhance its adaptability.

Infection and Immunity · Dec 13, 2021Peer-reviewedGenomics

Virulence phenotypes result from an interaction between pathogen ploidy and genetic background

Dorian J. Feistel, Rema Elmostafa, Meleah A. Hickman

The paper investigates how ploidy and genetic background of Candida albicans affect virulence in nematode hosts, finding significant interactions between ploidy and genetic background but no global virulence differences between diploid and tetraploid strains.

Ecology and Evolution · Aug 7, 2020Peer-reviewedGenetics

Ploidy determines the consequences of antifungal-induced mutagenesis in Candida albicans, a human fungal pathogen

Ognenka Avramovska, Meleah A. Hickman

This study shows that ploidy state in Candida albicans affects the mutation rates and types induced by antifungal drugs, with tetraploids showing higher mutagenesis under stress.

G3 Genes Genomes Genetics · Oct 5, 2019Peer-reviewedGenetics