Author

Stephanie Beate Geisler

Akershus University HospitalORCID 0009-0001-7191-6783Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Genetic & Genomic Medicine· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Approach:statistical model· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Assay:whole-exome sequencing· 1

Frequent Co-Authors

Jürgen GeisleriD· 2 shared Andliena TahiriiD· 1 shared Arvind Y.M. Sundaram· 1 shared Beryl Leirvaag· 1 shared Bjørnar GiljeiD· 1 shared Chandra L. TheesfeldiD· 1 shared Charles Joseph VaskeiD· 1 shared Denise G O'MahonyiD· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCancer Biology

Jürgen GeislerORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintSep 24, 2025

CLONAL SELECTION SUPPORTED BY SINGLE CELL DNA SEQUENCING REVEALS HORMONAL ADAPTATION AND RESISTANCE IN LOCALLY ADVANCED BREAST CANCER DURING NEOADJUVANT AROMATASE INHIBITION

Denise G O'Mahony, Tom Lesluyes, Ina Skaara Brorson…·Vessela Nedelcheva Kristensen

This study uses whole-exome sequencing and single-cell DNA sequencing to analyze subclonal evolution in breast cancer during aromatase inhibitor treatment, identifying resistance mechanisms and actionable targets.

DOI 10.1101/2025.09.24.678208

humanwhole-exome sequencing

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Genetic & Genomic MedicinemedRxivPreprintMay 14, 2023

Prenatal BRCA1 epimutations contribute significantly to triple-negative breast cancer development

Oleksii Nikolaienko, Hans Petter Eikesdal, Bjørnar Gilje…·Per Eystein Lønning

The paper identifies prenatal BRCA1 epimutations as a cause of approximately 20% of triple-negative breast cancers and low-ER expression breast cancers.

2 citations

humanDNA methylation

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