Cell BiologybioRxivPreprint
AMFR provides an ERAD bypass mechanism to maintain proteostasis under canonical E3 ligase deficiency
Uta Nakayamada, Aika Fujii, Saori Katoh…·Tsukasa Okiyoneda
The paper identifies AMFR as a compensatory factor for ERAD pathway function when RNF5/185 is deficient, showing its role in degrading mutant CFTR.
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