Author

A. Aguado

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:protein folding & dynamics· 2 Organism:bacteria (other)· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Muguruza-Montero· 2 shared A. Villarroel· 2 shared C. Malo· 2 shared E. Nunez· 2 shared J. Urrutia· 2 shared A. Bergara· 1 shared A. Leonardo· 1 shared C. Domene· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersCell Biology

A. Muguruza-Montero

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

SIEGA the integrated genomic surveillance system of Andalusia, a One Health regional resource connected with the clinic

C. S. Casimiro-Soriguer, J. Perez-Florido, E. A. Robles, M. Lara, A. Aguado, M. A. R. Iglesias, J. A. Lepe, F. Garcia, M. Perez-Alegre, E. Andujar, V. E. Jimenez, L. P. Camino, N. Lorusso, U. Ameyugo, I. M. Vazquez, , J. Dopazo

SIEGA is a genomic surveillance system that integrates data from humans, animals, and the environment to monitor infectious diseases and antimicrobial resistance.

bioRxiv · Feb 29, 2024 · PreprintMicrobiology

Redox regulation of Kv7 channels through EF3 hand of calmodulin

E. Nunez, F. Jones, A. Muguruza-Montero, J. Urrutia, A. Aguado, C. Malo, C. Domene, G. Bernardo-Seisdedos, O. Millet, N. Gamper, A. Villarroel

This study shows that EF3 hand of calmodulin mediates redox regulation of Kv7 channels by translating Ca2+ signals and reversing FRET signals in response to oxidation.

eLife · Feb 20, 2023Peer-reviewedCell Biology

AN EPILEPSY-CAUSING MUTATION LEADS TO CO-TRANSLATIONAL MISFOLDING

J. Urrutia, A. Aguado, C. Gomis-Perez, A. Muguruza-Montero, O. R. Ballesteros, J. Zhang, E. Nunez, C. Malo, H. J. Chung, A. Leonardo, A. Bergara, A. Villarroel

The paper shows that the W344R mutation in Kv7.2 channels causes co-translational misfolding, impairing CaM binding and leading to non-functional ion channels.

bioRxiv · Nov 25, 2020Biophysics